Skrevet av Emne: IKKE bruk oljer til steking!  (Lest 29170 ganger)

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Sv: IKKE bruk oljer til steking!
« #90 : 04. januar 2011, 23:53 »
Eller bare bruke heeelt enkelt.. vaaaanlig smør / kokosolje.

Føler ting blir gjort litt for komplisert her. Smiley

Bruk olivenolje om dere steker på lav temperatur, it's ok. Bruk smør om det er på høy temperatur, desto mer mettet fett desto bedre tåler fettet temperaturen :p

Men selv mettet fett gir etter en gang Smiley

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Sv: IKKE bruk oljer til steking!
« #91 : 08. januar 2011, 22:45 »
Blir vel litt som det som står på margarinet og alle de andre stivna planteoljene - "Et sunt alternativ til smør" o.l. Det står vel forøvrig også på formfettet man får kjøpt i butikken at det egner seg bra for steking, men har man respekt for helsen sin, egner det seg vel ikke som noe du bør putte inn i munnen din i det hele tatt. Bare fordi noe er markedsført som det ene eller det andre (f.eks sunt), betyr vel ikke det at det er det. Sier vel igrunn seg selv.

Det kan være en idé å sjekke fakta et annet sted enn på forpakningen til produktet når du kjøper mat.

Sier noe der.. Dog jeg har sjekket fakta andre steder også... Wink
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Utlogget Glucifer

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Sv: IKKE bruk oljer til steking!
« #92 : 08. januar 2011, 23:03 »
Olive oil stability under deep-frying conditions.
Food Chem Toxicol. 2010 Oct;48(10):2972-9. Epub 2010 Aug 3.
Casal S, Malheiro R, Sendas A, Oliveira BP, Pereira JA.

REQUIMTE/Serviço de Bromatologia, Faculdade de Farmácia da Universidade do Porto, Rua Aníbal Cunha 164, 4050-047 Porto, Portugal.
Abstract
The suitability of different commercial olive oil categories for domestic frying was investigated. Oil samples were taken every 3h of frying and evaluated for free acidity, peroxide and p-anisidine values, specific extinction coefficients, oxidative stability, fatty acids, vitamin E, β-carotene and total phenols, until the total polar compounds achieved the maximum legal value (25%). All olive oils were fried during more time than the commercial vegetable oil blend taken for comparison (from 24 to 27 h, against 15 h). The extra-virgin Protected Designation of Origin (PDO) olive oil was characterized by reduced levels of oxidation and hydrolysis, and superior amounts of minor antioxidant compounds. The "olive oil" commercial category behaves similarly, but "Cobrançosa" olive oils performance was slightly worse, and clearly different between years, highlighting the importance of blending different cultivars. The vegetable oil, despite containing significantly higher amounts of vitamin E, was highly susceptible to oxidation under frying conditions when compared to all olive oils. The results also show that the chemical composition of olive oils, particularly the amount of natural antioxidants, are important parameters in their predictive behavior along the frying process, but mostly that olive oil is clearly resistant to frying conditions, independently to the commercial category chosen.

Copyright (c) 2010 Elsevier Ltd. All rights reserved.
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Sv: IKKE bruk oljer til steking!
« #93 : 08. januar 2011, 23:06 »
Genotoxic and carcinogenic risks associated with the dietary consumption of repeatedly heated coconut oil.
Br J Nutr. 2010 Nov;104(9):1343-52. Epub 2010 Aug 6.
Srivastava S, Singh M, George J, Bhui K, Murari Saxena A, Shukla Y.

Proteomics Laboratory, Indian Institute of Toxicology Research (CSIR), Mahatma Gandhi Marg, Lucknow 226 001, UP, India.
Abstract
Repeated heating of vegetable oils at high temperatures during cooking is a very common cooking practice. Repeated heating of edible oils can generate a number of compounds, including polycyclic aromatic hydrocarbons (PAH), some of which have been reported to have carcinogenic potential. Consumption of these repeatedly heated oils can pose a serious health hazard. The objectives of the present study were to evaluate the genotoxic and carcinogenic risks associated with the consumption of repeatedly heated coconut oil (RCO), which is one of the commonly consumed cooking and frying medium. The PAH were analysed using HPLC in fresh CO, single-heated CO (SCO) and RCO. Results revealed the presence of certain PAH, known to possess carcinogenic potential, in RCO when compared with SCO. Oral intake of RCO in Wistar rats resulted in a significant induction of aberrant cells (P<0·05) and micronuclei (P<0·05) in a dose-dependent manner. Oxidative stress analysis showed a significant (P<0·05) decrease in the levels of antioxidant enzymes such as superoxide dismutase and catalase with a concurrent increase in reactive oxygen species and lipid peroxidation in the liver. In addition, RCO given alone and along with diethylnitrosamine for 12 weeks induced altered hepatic foci as noticed by alteration in positive (γ-glutamyl transpeptidase and glutathione-S-transferase) and negative (adenosine triphosphatase, alkaline phosphatase and glucose-6-phosphatase) hepatospecific biomarkers. A significant decrease in the relative and absolute hepatic weight of RCO-supplemented rats was recorded (P<0·05). In conclusion, dietary consumption of RCO can cause a genotoxic and preneoplastic change in the liver.

PMID: 20687968 [PubMed - indexed for MEDLINE]
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Sv: IKKE bruk oljer til steking!
« #94 : 08. januar 2011, 23:08 »
Genotoxic and Carcinogenic Risks Associated with the Consumption of Repeatedly Boiled Sunflower Oil.
J Agric Food Chem. 2010 Oct 1. [Epub ahead of print
Srivastava S, Singh M, George J, Bhui K, Shukla Y.

Proteomics Laboratory, Indian Institute of Toxicology Research (IITR), Council of Scientific and Industrial Research (CSIR), Post Office Box 80, Mahatma Gandhi Marg, Lucknow 226 001, India.
Abstract
Repeated boiling of vegetable oils at high temperature in cooking and frying is a very common practice and leads to the formation of a class of toxic substances. Among them, polycyclic aromatic hydrocarbons (PAHs) are well-documented for their mutagenic/carcinogenic potential. The objectives of the present study were to evaluate the genotoxic and carcinogenic risks associated with the consumption of repeatedly boiled sunflower oil, which is one of the commonly consumed vegetable oils in southeast Asian countries. The presence of PAHs was analyzed using high-performance liquid chromatography (HPLC) methods in fresh, single-boiled, and repeatedly-boiled sunflower oil (FSO, SBSO, and RBSO) samples. A higher amount of known carcinogenic/mutagenic PAHs in RBSO samples were shown, as compared to FSO and SBSO. Oral administration of RBSO in Wistar rats resulted in significant induction of aberrant cells (p < 0.05) and micronuclei (p < 0.05) incidence in a dose-dependent manner. Oxidative stress analysis also showed a significant decrease in levels of antioxidant enzymes, such as superoxide dismutase and catalase, with a concurrent increase in reactive oxygen species and lipid peroxidation in animals following RBSO consumption, as compared to FSO or SBSO (p < 0.05). Additionally, RBSO administration alone and along with diethylnitrosamine for 12 weeks induced altered hepatic foci, as noticed by the alteration in positive (γ-glutamyl transpeptidase and glutathione-S-transferase) and negative (adenosine-triphosphatase, alkaline phosphatase, and glucose-6-phosphatase) liver biomarkers. A significant decrease in the relative and absolute hepatic weight in RBSO-supplemented rats was also noted (p < 0.05).

PMID: 20886885 [PubMed - as supplied by publisher]
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